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Unfortunately women's health clinic unionville buy 70 mg fosamax with visa, the relapsing nature of the disease means long-term treatment may be needed with oral corticosteroids womens health boulder fosamax 70 mg sale, with the inevitable risk of side effects. This may be related to the fact that healthy gastric and intestinal mucosa harbors eosinophils under physiological conditions and so it is difficult to establish "normal" cell numbers. During the 70 years since it was first described by Kaijser in 1937,19a approximately 200 cases have been reported in the literature. Mucosal involvement is typically associated with vomiting, diarrhea, abdominal pain, weight loss, failure to thrive, and either occult or frank bleeding. Involvement of the muscular layers may lead to signs and symptoms of intestinal obstruction, whereas patients with serosal involvement typically complain of bloating and can present with ascites. Throughout the course of the disease, the clinical manifestations vary according to which organs are involved. During its long-term course, extraintestinal manifestations or lymphoproliferative conditions may occur and facilitate the definite diagnosis. Intestinal manifestations and cardiac involvement are associated with poor prognosis and risk of fatal outcome, whereas patients with skin disease generally have a milder course. The findings are usually not spectacular and include thickening of the intestinal folds with deformation of the luminal configuration, diminished peristalsis, and an erythematous and friable mucosa. Moreover, endoscopy enables representative biopsy samples to be taken for histological confirmation of the diagnosis. Diagnostic confirmation is hampered by the difficulty in obtaining histological samples. Cytotoxic agents, such as hydroxyurea, vincristine, and chlorambucil, have been successfully used in corticosteroid-refractory patients. Prognosis depends on the degree and location of the eosinophil-mediated end-organ tissue damage. The physiological and pathophysiological roles of eosinophils in the gastrointestinal tract. Eosinophilic esophagitis: updated consensus recommendations for children and adults. Eosinophilic gastroenteritis: a clinicopathological study of patients with disease of the mucosa, muscle layer, and subserosal tissues. A striking local esophageal cytokine expression profile in eosinophilic esophagitis. Eotaxin-3 and a uniquely conserved gene-expression profile in eosinophilic esophagitis. Eosinophilic esophagitis is characterized by a non-IgE-mediated food hypersensitivity. Idiopathic eosinophilic esophagitis is associated with a T(H)2-type allergic inflammatory response. Natural history of primary eosinophilic esophagitis: a follow-up of 30 adult patients for up to 11. Development and validation of a symptom-based activity index for adults with eosinophilic esophagitis. Endoscopic assessment of the oesophageal features of eosinophilic oesophagitis: validation of a novel classification and grading system. Zur Kenntnis der allegischen Affektioner desima Verdauungskanal von Standpunkt desima Chirurgen aus. Patients, usually males, present with recurrent dysphagia and food impaction as well as coexisting allergic airway diseases. Untreated EoE can induce structural changes of the esophagus, eventually leading to an extremely fragile and rigid esophageal wall structure with increased risk for complications, including perforation. Effective treatments for adult patients with EoE include swallowed topical (or, seldom, systemic) corticosteroids, hypoallergenic diets, and esophageal dilatation. Though quality of life is substantially diminished in this chronic disorder, life expectancy is not affected. Mucosal involvement, the most frequent subtype, is typically associated with vomiting, diarrhea, abdominal pain, and weight loss. Patients with the serosal form complain mainly of bloating and ascites and usually respond dramatically to corticosteroids. Use of an anti-interleukin-5 antibody in the hypereosinophilic syndrome with eosinophilic dermatitis. Interleukin 5 and phenotypically altered eosinophils in the blood of patients with the idiopathic hypereosinophilic syndrome. During the natural course of the disease, often involves stomach and small bowel C. Immunoglobulin E (IgE)-based testing plays a crucial role in the identification of causative foods 2. Dilatation is regarded as first-line treatment due to the rapid symptom resolution D. Require a comprehensive hematological and multisystem examination 47 Allergic Disorders of the Eye Virginia L. Research to identify the inflammatory cells and molecules involved in allergic responses at the ocular surface and their role in the pathogenesis of allergic eye diseases has increased in the past few years. Extensive immunohistochemical studies have identified distinct expression profiles of adhesion molecules, chemokine receptors, cytokines, and immune cell types for each subtype of allergic conjunctivitis, while immune pathways are being studied via novel experimental models. This article reviews recent advances in our understanding of the cellular and molecular processes occurring in ocular allergy.

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Unstimulated tissue mast cells continually secrete immature protryptase into the tissue womens health partners boca raton buy generic fosamax 35 mg on line, and it diffuses into the circulation to provide a measure of total mast-cell number menstrual irregularities and thyroid fosamax 70 mg buy mastercard. Mature tryptase is normally undetectable (<1 µg/L) in serum from healthy individuals who have no history of anaphylaxis during the preceding hours. Baseline levels of >20 µg/L are detected in most individuals with systemic mastocytosis. Allergen-Specific IgG Allergen immunotherapy is known to enhance the production of specific IgG "blocking" antibodies. However, clinically successful immunotherapy is almost always accompanied by high serum levels of allergen-specific IgG (typically IgG1 and IgG4) blocking antibodies. Its utility in the diagnosis of sensitization to other allergen specificities appears more promising, but further documentation involving clinical studies is required. Recommended serum collection times for tryptase quantification range from 30 minutes to 4 hours after the onset of an acute event. Because serological tests for mature tryptase are not widely available, it is important to compare an acute event total tryptase level (within 4 hours) with a baseline total tryptase 24 hours after all signs and symptoms of the event have subsided. However, reported mature tryptase levels in postmortem cases of fatal anaphylaxis have ranged from 12 to 150 µg/L in all nine fatalities caused by Hymenoptera venom and in six of eight food-induced fatalities. A variety of criteria are used to identify an allergen-induced positive response, such as a stimulation index (allergen-induced/basal ratio) >2. Its performance was evaluated by using minor determinant mixture, benzyl penicilloyl polylysine, penicillin, ampicillin, amoxicillin, and cephalosporin as stimulating antigens. Diagnostic sensitivity was marginally increased to 66% by simultaneously using the drug-specific IgE antibody serology result. Additionally, there is no need for the use of an additional fluorescent anti-IgE reagent to gate the basophils. Some assays monitor the magnitude of mediator release from basophils, and others assess the degree of cell surface marker expression following exposure to allergen. Histamine Release Assay the potent vasoactive mediator histamine is stored in cytoplasmic granules of basophilic leukocytes and mast cells. It is released along with other mediators of inflammation in response to both immunological and nonimmunological stimuli. In its most basic form, peripheral blood leukocytes are isolated from a donor and incubated with varying concentrations. Histamine release is complete within 30 minutes, and then histamine in the supernatant is measured by enzymatic, radiometric, or spectrophotofluorometric techniques. The results are highly correlated with those determined by skin testing71 and bronchoprovocation. One false-positive result was observed among 13 healthy controls with a negative result on the skin test. Diagnostic Utility of Basophil Activation Flow Cytometric Assays the utility of basophil activation flow cytometric assays is limited by a number of technical concerns. Third, varying concentrations of crude allergen extract or recombinant allergen or anti-IgE are incubated with the cell preparation. Thus each stimulating allergen needs to be prequalified with basophils from well-characterized patients before use. Fourth, criteria for defining positive assay results vary among the laboratories performing the test, and stimulating allergen preparations from even the same manufacturer can have variable potency. Possibly most important, when the performance characteristics were directly compared in clinical studies to puncture skin test results, the diagnostic sensitivity and specificity of the flow-cytometric analysis of in vitro allergen-activated basophils was less than optimal. For these reasons, cell-based cytometric methods will most likely remain useful research techniques for the investigation of allergic disease, but they will have limited application to clinical diagnosis. Association of reaginic activity with an immunoglobulin other than gamma A or gamma G globulin. Distribution of IgE in a community population sample: correlations with age, sex and allergen skin test reactivity. Age-related serum IgE levels in healthy subjects and in patients with allergic disease. Efficacy and safety of omalizumab in patients with chronic idiopathic/spontaneous urticaria who remain symptomatic on H1 antihistamines: a randomized, placebo-controlled study. Clinical history-driven diagnosis of allergic diseases: utilizing in vitro IgE testing. Allergic sensitization is a key risk factor for but not synonymous with allergic disease. In vivo methods for study and diagnosis of allergy: skin tests, techniques and interpretation. The liberation of a histamine-like substance in injured skin, the underlying cause of factitious urticaria and of wheals produced by burning; and observations upon the nervous control of certain skin reactions. Performance and pain tolerability of current diagnostic allergy skin prick test devices. The 3rd international standard for serum IgE: international collaborative study to evaluate a candidate preparation. Technological innovations for high-throughput approaches to in vitro allergy diagnosis. Clinical laboratories worldwide need to report IgE antibody results on clinical specimens as analytical results and not use differential positive thresholds. Responsibility for quality IgE antibody results rests ultimately with the referring physician.

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However menopause reset fosamax 35 mg order with visa, each of these conditions has different properties and probably has its own pathogenesis; to subsume them into one single category is quite arbitrary and based exclusively on descriptive features pregnancy stages week by week cheap fosamax 70 mg buy on-line. EoE is definitely an esophageal-restricted disease with a benign long-term prognosis. Because of its clinical relevance, EoE is discussed in greater depth here than the other two eosinophil-associated disorders. Epidemiology Epidemiological information is essential to determine the clinical and socioeconomic impact of a disease. Therefore knowledge of the epidemiological parameters of a disease is crucial for identifying risk factors as well as pathogenetic mechanisms, for planning preventive measures, and for determining optimal treatment approaches. Demographic Cornerstones Cases of EoE have been reported worldwide and identified in those with a variety of ethnic backgrounds, including Caucasians, African Americans, Hispanics, and Asians. However, as there are no controlled data about geographical variations of prevalence, it remains unclear whether EoE is associated with any particular 633 634 Part five Allergic Diseases incidence and prevalence of EoE in the adult population during recent years. Between 1989 and 2009, EoE was diagnosed in 46 patients (76% males; mean age 41 ± 16 years). In the face of a constant diagnostic delay and the lack of EoE awareness programs, significantly more EoE cases were diagnosed from 2000­2009 compared with 1989­1999. Because EoE is clinically a benign disease affecting mainly younger individuals, the cumulative EoE prevalence rose to 42. Extrapolating from this study, in North America and in Europe there is one patient with diagnosed EoE among every 2­3000 inhabitants. EoE can be found in all age groups, but most studies report an average age between 34 and 42 years with a male-to-female risk ratio of 3: 1. Interestingly, age at diagnosis does not correlate with onset of EoE-attributed symptoms, which can be considered as the onset of disease. Often patients develop specific eating strategies, with careful chewing and avoidance of dry and rough food; despite a substantial impairment of quality of life, they bypass medical care (patient delay). An average diagnostic delay of 5 to 6 years has been reported from both North America and Europe. On the other hand, clinical trials of targeted food elimination diets, or treatment with IgE-blocking agents, have failed to show an IgE-mediated mechanism. In summary, the current understanding is that IgE plays at most a subsidiary role in the pathogenesis of EoE and that neither IgE-based diagnostic tools nor IgE-targeted treatment is helpful. The current understanding of cellular and molecular mechanisms in the pathogenesis of eosinophilic esophagitis (EoE) involves a complex interaction of genetic and environmental factors. Two possible pathways can lead to accumulation of eosinophils in the esophageal wall. Representative endoscopic pictures of active eosinophilic esophagitis showing loss of vascularity and red furrows (A), whitish exudates (B), corrugated rings (C), and a long-distance stricture with a deep laceration after dilatation (D). In contrast, food refusal and failure to thrive may be observed in children, as dysphagia is less easily expressed in this age group. Short-term use of systemic corticosteroids may be limited to emergent cases, such as dysphagia requiring hospitalization, patients with dehydration due to swallowing difficulties, or patients with symptoms refractory to topical steroids. Discontinuation of topical and systemic corticosteroids is usually followed by recurrence of the disease within a few weeks. Cromolyn sodium has no apparent therapeutic effect, and although leukotriene receptor antagonists have been shown to induce symptomatic relief, they do not affect esophageal eosinophilia. Only limited data are available for targeted therapy with novel biological agents or immunosuppressants. The immunosuppressants azathioprine and 6-mercaptopurine were effective in inducing and maintaining remission in three corticosteroid-refractory EoE patients. However, further evaluation of these alternatives for corticosteroid-refractory EoE is needed before they can be implemented into daily clinical practice. Diet Several prospective uncontrolled trials have been conducted to assess the potential of three different diets in treating EoE. Individually tailored so-called targeted elimination diets, empirical six-food elimination diets (removal of the six most common allergenic foods such as dairy, eggs, wheat, soy, peanuts, fish/ shellfish), and a protein-free elemental diet have all shown efficacy in active EoE. Overall, the value and the feasibility of dietary therapy require further evaluation. Dilatation Esophageal dilatation leads to long-lasting symptom relief but does not influence the underlying inflammation. It should therefore be reserved for patients who present with functional esophageal narrowing (strictures, stenosis) that is refractory to drug therapy. Endoscopic dilatation should be performed with caution, as it carries a risk of esophageal injury, although recently fiG 46. Representative histological pictures of active eosinophilic esophagitis showing a dense infiltration of the squamous epithelium with eosinophils forming microabscesses. Eversion of the lids to reveal the tarsal conjunctiva demonstrates some hyperemia and mild infiltration (loss of transparency and thickening) of the conjunctiva with diffuse small inflammatory excrescences known as papillae. Because there is no serious limbal disease or conjunctival scarring and the cornea is not involved, the visual acuity remains normal. This pattern of expression is correlated with the degree of neutrophil or eosinophil infiltration in the bulbar tissue, suggesting a mast-cell­mediated cell recruitment process. The disorder can occur at any age but is more frequently seen in children and young adults and tends to lessen in severity in the older age groups. Patients present with seasonally recurring itching, watering, redness, and swelling of the eyes and lids, and there may be increased conjunctival discharge. During the allergen season, the ocular signs range from mild to dramatic and are usually bilateral and symmetrical.

Syndromes

• Stopping or changing the doses of medicines you are taking, such as ACE inhibitors, angiotensin receptor blockers, spironolactone (Aldactone), amiloride (Midamor), or triamterene (Dyrenium)
• Blood
• Pregnancy
• Dizziness
• Doing household chores (sweeping, mopping, and vacuuming floors, loading the dishwasher)
• Dopamine blockers (haloperidol, quetiapine, or risperidone are most commonly used)
• Normal peak value -- at least 10 ng/mL
• Skin sores, which may become a skin ulcer that heals very slowly
• An electroencephalogram (EEG) may be used to rule out epilepsy as a cause of the apraxia.
• Yawning

Monoclonal antibodies specific for human IgE producing B cells: a potential therapeutic for IgE mediated allergic diseases womens health kettlebell fosamax 35 mg buy low price. Evaluation of two commercial omalizumab/free IgE immunoassays: implications for use during therapy pregnancy pillows purchase discount fosamax. Monitoring allergic patients on omalizumab with free and total serum IgE measurements. Allergens are distributed into few protein families and possess a restricted number of biochemical functions. Allergy work-up including component-resolved diagnosis: how to make allergen-specific immunotherapy more specific. Tryptase levels as an indication of mast cell activation in a patient with Hymenoptera anaphylaxis and mastocytosis. Correlation of skin test results with in vitro whole blood histamine release in 82 patients. An evaluation of spontaneous histamine release and the low responders in a basophil histamine release test. Superiority of the histamine release test above case history, prick test and radioallergosorbent test in predicting bronchial reactivity to the house dust mite in asthmatic children. Mononuclear blood cell sulpholeukotriene generation in the presence of interleukin 3 and whole 1295 76. Studies of cell adhesion and flow cytometric analyses of degranulation, surface phenotype and viability using human eosinophils, basophils and mast cells. In vitro diagnosis of immediate drug hypersensitivity: should we go with the flow The basophil activation marker defined by antigen 97A6 is identical to ectonucleotide pyrophosphate/ phosphodiesterase 3. The clinical utility of basophil activation testing in diagnosis and monitoring of allergic disease. In the diagnostic workup of a patient with a clinical history of a systemic anaphylactic reaction involving hypotension following a sting from a Hymenoptera, total and mature mast cell tryptase were requested to confirm mast cell involvement. What is the correct time to collect a whole blood specimen for a total and mature tryptase analysis In the workup of a patient for a food allergy, several procedures are typically involved. Oral provocation tests are first performed, followed by the clinical history and then in vivo and/or in vitro tests for immunoglobulin E (IgE) antibody (sensitization). In vitro tests for sensitization are performed first, followed by collection of a clinical history and then oral provocation testing. Clinical history, followed by either in vivo or in vitro tests for sensitization and then an oral provocation test, if needed. Performing a multiplex chip­based IgE antibody assay for identifying sensitization before collecting a clinical history from the patient. Exposure to which of the following principal allergen families poses the most serious risk for the induction of a systemic allergic reaction in an individual with peanut allergy Storage proteins (2S albumins, 7S globulins [vicilins], and 11S globulins [legumins]), which are involved in nutrient management D. Tropomyosins, which are integral components of actin filaments 96 Molecular Methods John W. Belmont Molecular analysis of genes and genomes can be central to arriving at a precision diagnosis. A few of these apparently act as "master" genes during particular developmental processes or in specific cell lineages. So far, immunodeficiency genes appear to be in the category of genes that involve either innate or adaptive immunity controlling cell growth, differentiation, effector functions, or apoptosis. Protein and nucleotide differences are called polymorphisms when they are frequent enough to be found in 1­5% of the general population. Variations in single nucleotides occur in 1/100 bases when whole-genome sequencing is used to survey individuals. Some polymorphisms involve simple sequence repeats so that there is variation in the number of repeat units. The composition of alleles within genetic loci in an individual is called the genotype. Some components of the phenotype, such as body weight, are simple to measure, whereas other clinically important phenotypes are based on complex laboratory evaluation. A key distinction is drawn between discrete traits (normal vs abnormal) and quantitative traits (continuous range of values). Polymorphisms account for some of the variation observed at the phenotype level between healthy individuals or between populations, and the cumulative percentage of the variation explained by genetic variation is called heritability. In monogenic diseases (also called mendelian diseases), the presence of mutation(s) is usually considered necessary and sufficient to cause disease. The aggregate protein coding sequences, referred to as the "exome," account for about 1­1. These repeat elements are generally silent, but they may be involved in some types of gene regulation and can become involved in mutational mechanisms of deletion, duplication, and insertion. Other genes are under very specific control, and their expression is restricted to one or a few cell types. Structural variants have been found to be a common cause of human genetic disorders. Linkage mapping played a large role in the identification of the X-linked immunodeficiency genes (Chapters 22, 33­36). This initial arrangement of alleles, however, is broken up by recombination over time. It is also apparent that the extent of individual genetic variation was underestimated from previous data and that each individual bears about 3.

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Recent research suggests that glial cells also play a role in a number of cellular processes women's health evergreen order generic fosamax from india, such as facilitating communication among neurons women's health center waterbury ct buy fosamax with mastercard, plasticity, and production of substances that nourish neurons and enhance their survival (neurotrophins; Otten et al. Glial cells are subdivided into two main classifications: macroglia and microglia. Macroglia are the larger glial cells, and in the brain, they include astrocytes and oligodendrocytes. Astrocytes are the largest and most abundant type of glial cell and provide structural support for neurons. In addition, they are in physical contact with blood vessels and form a protective barrier so toxic substances do not penetrate the brain. Evidence suggests that astrocytes are involved in the production of chemicals that neurons use to communicate with each other and that the interaction between astrocytes and neurons may be impaired during degenerative brain diseases such as dementia (Hertz et al. Evidence also suggests that astrocytes react to injury of the brain in a number of ways, including the formation of scar tissue that serves to reduce further inflammation of the brain but also interferes with axonal regeneration. Interestingly, the pattern of astroglial scarring in the brain appears to differ depending on the source of the brain injury. For example, preliminary findings suggest that military personnel suffering Neuroanatomy, Brain Development Table 1. Glial cells are categorized into two types: smaller cells known as microglia and larger cells known as macroglia. Astrocytes perform a variety of functions, including providing nutrients to neurons and removal of waste products, providing support to cells that form the blood-brain barrier, and releasing neurotrophins and neuromodulators. Oligodendrocytes provide structural support for neurons and insulate axons by ensheathing axons with concentric layers of myelin. Myelin consists of ~80% lipids and 20% protein, and facilitates the rapid transduction action potentials down the axon. Microglial cells play a major role in phagocytosis, as microglia are scavengers that remove waste and debris, including dead neurons. Macroglia: Astrocytes Macroglia: Oligodendrocytes Microglia Cells brain injury as a result of blast exposure have a particular pattern of astroglial scarring that may predict psychological outcomes such as post-traumatic stress disorder (Shively et al. Research also suggests that astrocytes may strengthen signaling among neurons by releasing substances that amplify the effects of neurotransmitters (Do et al. In contrast to the traditional view of glial cells as exclusively "nurse cells", current research suggests that glial cells often serve to modulate neurotransmission and are also capable of releasing neurotransmitters that activate receptors on neurons, other glial cells, and vascular cells (Bellot-Saez et al. A second type of macroglial cell, oligodendrocytes form a protective sheath, known as myelin, around part of the neuron: the axon. Myelin consists of lipids and proteins, and helps to insulate and facilitate the transduction of messages sent from one neuron to another. The remaining 30% is found in the membranes of glial cells and neurons where it is used for neuronal repair. A single oligodendrocyte can myelinate multiple neurons via extensions that wrap around the axon of neurons. Recent findings suggest that microglia may play a role in neurodegenerative disease, epilepsy, cerebral palsy, and autistic spectrum disorder due to their release of substances that are associated with inflammation of brain tissue. In summary, glial cells (a) participate in the uptake and breakdown of chemicals that neurons use for communication; (b) act as scavengers and remove waste products and debris, including dead neurons; (c) take up ions from the extracellular environment; (d) provide proteins and other substances to neurons; (e) segregate groups of neurons from one another; and (f) modulate neuronal signaling (Levitan & Kaczmarek, 1997, p. Neurons Neurons come in different shapes, sizes, and types, and are located differentially throughout the brain. As reviewed by Thompson (2000), there are four main functional classifications of neurons: motor, sensory, principal, and interneurons. Motor neurons have axons that project to the spinal cord where they communicate with other motor neurons that innervate muscles and glands. Principal neurons have long axons that extend to other locations in the brain, whereas interneurons, the most abundant neurons, have shorter axons that communicate messages to nearby cells and convey information between sensory and motor neurons. The role of interneurons in epilepsy, addiction, and other human conditions is also being studied (Librizzi et al. The basic components of a neuron include a cell body (soma), dendrites, and an axon. The cell body contains the nucleus and other structures that help a neuron process and transmit information. Dendrites are very thin structures that emerge from the cell body of the neuron and are formed in a variety of shapes and sizes. They receive messages from other neurons while axons transmit messages to other cells. Dendrites continue to develop and mature postnatally, and are believed to play an integral role in neuroplasticity (Kennedy, 2000; Chen et al. Axons of neurons extend from the cell body and transmit information via the terminal button to other neurons, a process known as neurotransmission. Internal components of neurons and the process of neurotransmission are covered in detail in the Chapter 2. Neuroblasts are immature neurons and glioblasts are immature glial cells; the formation of these cells is known as neurogenesis and gliogenesis, respectively. Factors that regulate the processes of neurogenesis and gliogenesis are not well understood, but neuroscientists are studying the role of genes and various proteins that trigger cellular change and maturation. In recent years, a number of factors have been identified, including proteins that promote the division of neural stem cells, neurotrophic substances. This continues to be an active area of research in which future studies will likely reveal the complexities involved in the formation of neurons and glial cells.

References

• Sinnreich M, Klein CJ, Daube JR, Engelstad J, Spinner RJ, Dyck PJ. Chronic immune sensory polyradiculopathy. Neurology. 2004;63:1662-1669.
• Hediger EM, Gold H. U.S.P ether from large drums and ether from small cans labeled 'for anesthesia'. JAMA. 1935;104:2244-2248.
• Lopate G, Parks BJ, Goldstein JM, Yee WC, Friesenhahn GM, Pestronk A. Polyneuropathies associated with high titre antisulfatide antibodies: characteristics of patients with and without serum monoclonal proteins. J Neurol Neurosurg Psychiatry. 1997;62:581-585.
• Lughezzani G, Jeldres C, Isbarn H, et al. A critical appraisal of the value of lymph node dissection at nephroureterectomy for upper tract urothelial carcinoma. Urology 2010;75(1):118-124.
• Lucey MR, Mathurin P, Morgan TR. Alcoholic hepatitis. N Engl J Med 2009;360(26):2758-69.
• Fabre O, Vincentelli A, Willoteaux S, et al: Preoperative fenestration for type A acute aortic dissection with mesenteric malperfusion, Ann Thorac Surg 73(3):950-951, 2002.
• Chimenti C, Pieroni M, Morgante E, Antuzzi D, Russo A, Russo MA, et al. Prevalence of Fabry disease in female patients with late-onset hypertrophic cardiomyopathy. Circulation. 2004;110:1047-53.