Dipyridamole

Dipyridamole 100mg

  • 30 pills - $26.28
  • 60 pills - $42.81
  • 90 pills - $59.35
  • 180 pills - $108.94
  • 270 pills - $158.54
  • 360 pills - $208.14

Dipyridamole 25mg

  • 60 pills - $28.08
  • 90 pills - $34.71
  • 120 pills - $41.33
  • 180 pills - $54.59
  • 270 pills - $74.47
  • 360 pills - $94.35

Immunogenicity and safety of recombinant rabies viruses used for oral vaccination of stray dogs and wildlife arteria 66 25 mg dipyridamole order. Rabies post-exposure prophylaxis for a child with severe allergic reaction to rabies vaccine blood pressure medication new zealand dipyridamole 25 mg buy low price. Era vaccinederived cases of rabies in wildlife and domestic animals in Ontario, Canada, 1989-2004. Assessment of inactivated human rabies vaccines: Biochemical u characterization and genetic identification of virus strains. Human diploid cell rabies vaccine purified by zonal centrifugation: A controlled study of antibody response and side effects following primary and booster pre-exposure immunizations. Risk factors for systemic hypersensitivity reactions after booster vaccinations with human diploid cell rabies vaccine: A nationwide prospective study. Higher production of rabies virus in serum-free medium cell cultures on microcarriers. World Health Organization attitude concerning the use of continuous cell lines as substrates for production of human virus vaccines. Laboratory data supporting the clinical trial of anti-rabies serum in persons bitten by a rabid wolf. High vero cell density and rabies virus proliferation on fibracel disks versus cytodex-1 in spinner flask. Replacement, reduction and refinement alternatives to animal use in vaccine potency measurement. Analysis on the risks of severe adverse events in rabies post-exposure prophylaxis and appropriate decision-making procedure. Three-year duration of immunity in cats vaccinated with a canarypox-vectored recombinant rabiesvirus vaccine. Immunogenicity and efficacy of an in-house developed cell-culture derived veterinarian rabies vaccine. Studies on chick embryo adapted rabies virus; culture characteristics and pathogenicity. Development of a new purified vero cell rabies vaccine (Rabivax-S) at the serum institute of India Pvt Ltd. Evaluation of the safety, immunogenicity, and pharmacokinetic profile of a new, highly purified, heat-treated equine rabies immunoglobulin, administered either alone or in association with a purified, Vero-cell rabies vaccine. A next-generation, serum-free, highly purified Vero cell rabies vaccine is safe and as immunogenic as the reference vaccine Verorab when administered according to a post-exposure regimen in healthy children and adults in China. Characteristics and viral propagation properties of a new human diploid cell line, Walvax-2, and its suitability as a candidate cell substrate for vaccine production. Intradermal vaccination for rabies prophylaxis: Conceptualization, evolution, present status and future. Effect of preexisting immunity to adenovirus human serotype 5 antigens on the immune responses of nonhuman primates to vaccine regimens based on human- or chimpanzee-derived adenovirus vectors. Validation of the inactivant binary ethylenimine for inactivating rabies virus for veterinary rabies vaccine production. Development and prevalidation of a quantitative multi-dose serological assay for potency testing of inactivated rabies vaccines for human use. An acute neurologic syndrome temporally associated with postexposure treatment of rabies. Mechanistic actions of the risks and adverse events associated with vaccine administration. Analysis of vaccine-virus-associated rabies cases in red foxes (Vulpes vulpes) after oral rabies vaccination campaigns in Germany and Austria. Experiences with the aerial distribution of baits for the oral immunization of foxes against rabies in eastern Germany. Studies with human diploid cell strain rabies vaccine and human antirabies immunoglobulin in man. An epidemic of sylvatic rabies in Finland-Descriptive epidemiology and results of oral vaccination. IgE reactivity to vaccine components in dogs that developed immediate-type allergic reactions after vaccination. Manual of diagnostic tests and vaccines for terrestrial animals (mammals, birds and bees) (7th ed. A novel canine favored CpG oligodeoxynucleotide capable of enhancing the efficacy of an inactivated aluminum-adjuvanted rabies vaccine of dog use. Trap-vaccinate-release and oral vaccination for rabies control in urban skunks, raccoons and foxes. Use of a reduced (4-dose) vaccine schedule for postexposure prophylaxis to prevent human rabies: Recommendations of the advisory committee on immunization practices. Oral vaccination of raccoons (procyon-lotor) with an attenuated (Sad-B19) rabies virus-vaccine. Oral vaccination of wildlife against rabies: Opportunities and challenges in prevention and control. Oral immunization and protection of raccoons (Procyon lotor) with a vaccinia-rabies glycoprotein recombinant virus vaccine. Proceedings of the National Academy of Sciences of the United States of America, 83(20), 7947­7950. The rabies early death phenomenon: A report of ineffective administration of rabies vaccine during symptomatic disease. IgE reactivity to alpha1 and alpha2 chains of bovine type 1 collagen in children with bovine gelatin allergy.

Dipyridamole dosages: 100 mg, 25 mg
Dipyridamole packs: 30 pills, 60 pills, 90 pills, 180 pills, 270 pills, 360 pills, 120 pills

The factors responsible for the resistance of the kidney to bacterial invasion are not known blood pressure treatment dipyridamole 25 mg low price. However blood pressure chart normal blood pressure range purchase discount dipyridamole line, the susceptibility to colonization and infection is increased if there are focal areas of hypoperfusion or incomplete or partial obstruction of the excretory system. The normal urinary tract is sterile and ascending infections begin with the colonization by these bacteria not only of the periurethral area but also of the urethra and then the bladder. In the absence of an anatomic lesion (such as prostatic hypertrophy), bladder infections (cystitis) occur primarily in females. The enhanced susceptibility of females is due to three factors: the presence of a vaginal reservoir of bacteria, such as Escherichia coli, derived from the fecal flora the short female urethra, which promotes growth of colonies and passage of bacteria into the bladder, particularly as a consequence of intercourse the lack of prostatic fluid with antibacterial properties the net effect is that the incidence of urinary tract infection is 3% to 5% in normal women during the reproductive age, approximately 50 times higher than that in normal young men. Urinary tract infections are more common in elderly patients and almost as likely to occur in men as in women in this age group. Prostatic disease is an important risk factor in older men because the prostate can act as a nidus of infection, and incomplete emptying of the bladder due to partial urethral obstruction can prevent bacterial elimination by voiding. In addition to gender, both bacterial and host factors are important determinants of infection by increasing adhesion of bacteria to the uroepithelial cells; in the absence of adhesion, bacterial excretion during urination is likely to occur. As an example, almost all bacteria that cause acute pyelonephritis in otherwise healthy women have Gal-Gal pili that attach to digalactoside receptors contained in glycolipids on the surface of uroepithelial cells; these glycolipids are part of the P blood group antigen. This response is in part genetically determined, as the cell surface glycolipids from these patients more avidly bind E. Once bladder infection has occurred, vesicoureteral reflux plays a central role in the development of ascending pyelonephritis. Under normal circumstances, the most distal portion of the ureter is located within the bladder wall and traverses it at an angle. This intramural segment of the ureter is compressed when the pressure inside the bladder increases during micturition, resulting in an effective valve-like mechanism that prevents the retrograde flux of urine. This protective response is lost when the vesicoureteral junction is distorted due to bladder infection or to a congenital malformation often associated with a shortened intramural segment. In this setting, increased bladder pressure during urination results in the movement of infected urine into the ureter and the renal pelvis. The development of pyelonephritis requires an additional step: intrarenal reflux of infected urine into the kidney parenchyma. During embryonic development in higher mammals, several lobes of parenchyma can fuse, resulting in compound papillae; this is most likely to occur at the upper and lower poles. The ducts of Bellini that end at the concave portion of a compound papilla do so through circular, relatively open orifices, particularly in children under age 7 years. Thus, increased intrapelvic pressure transmitted upstream by vesicoureteral reflux generates a force that promotes reflux of urine into the parenchyma at these sites. On a simple conical papilla, shown on the left, the ducts of Bellini have slender slit-like openings. On a compound papilla, shown on the right, an increase in pressure (as occurs during micturition in patients with vesicoureteral reflux) results in forces perpendicular to the surface of the papilla; orifices on the convex surface act as valves and tend to close while those that open on the concave portion are distended, a process that allows urine and bacteria to be propelled into the kidney parenchyma. Involvement of the lower urinary tract (urethra and bladder) is confined mostly to the superficial layers of the mucosa, and significant tissue invasion does not usually occur. As a result, the primary symptoms are local discomfort on urination (dysuria) and urinary frequency and urgency; signs of systemic infection, such as fever and malaise, are absent. Examination of the urine sediment typically shows bacteria and white cells (pyuria). Thus, affected patients typically complain of pain and tenderness over the kidney, fever, and chills. Bacteriuria and pyuria are again present (unless there is infection behind a completely obstructed kidney); white cell casts, if seen, confirm kidney involvement. Given the differing pathogeneses of hematogenous and ascending pyelonephritis, the presence or absence of which symptoms might help to distinguish clinically between these disorders Pathology the distribution of lesions within the kidney in acute pyelonephritis is somewhat unpredictable, although the upper and lower poles with compound papillae are more frequently involved in ascending infection. The initial lesion is characterized by interstitial edema and neutrophilic infiltration. The inflammatory process in hematogenous pyelonephritis soon involves the tubules and spreads into the medullary segments of the nephron, where large collections of neutrophils can be seen filling the collecting ducts. In the presence of severe infection or delayed antimicrobial therapy, there is eventual destruction of the kidney parenchyma and the formation of irregular abscesses and eventually scars. For example, diabetic vascular disease or the increased intrarenal pressure induced by urinary tract obstruction can lead to a critical reduction in medullary blood flow that, in the presence of infection, can induce papillary necrosis. Healing in the kidney after resolution of the infection occurs through transformation of the neutrophil-rich exudate into active granulation tissue. Scarring is more prominent in children with congenital vesicoureteral reflux who, as noted earlier, are also more likely to have intrarenal reflux due to open orifices of the ducts of Bellini present in the compound papillae. It is not always easy by imaging techniques to distinguish pyelonephritic scars from ischemic lesions or healed infarcts. Pyelonephritic scars characteristically affect the renal poles where compound papillae occur. There is atrophy of the cortical elements, which results in an irregular indentation of the surface of the kidney (arrows). The underlying tissue is scarred and the calix that drains such a papilla is deformed.

Ampalaya (Bitter Melon). Dipyridamole.

  • Are there any interactions with medications?
  • Dosing considerations for Bitter Melon.
  • How does Bitter Melon work?
  • Diabetes, a skin condition called psoriasis, HIV/AIDS, stomach and intestinal disorders such as ulcers and constipation, kidney stones, liver disease, and skin abscesses and wounds.
  • What is Bitter Melon?
  • Are there safety concerns?

Source: http://www.rxlist.com/script/main/art.asp?articlekey=96773

The renal failure in accidental haemorrhage complicating pre-eclampsia is more a result of tubular necrosis and less of cortical necrosis blood pressure in the morning order dipyridamole visa. If prompt and adequate blood transfusions are given to these subjects prehypertension in late pregnancy 25 mg dipyridamole order with mastercard, renal failure can be reversed or altogether prevented. It is relevant to note that accidental haemorrhage has a notorious habit to recur and also has the complication of renal shut down. Nevertheless, with better monitoring facilities available and better neonatal intensive care, obstetricians tend to intervene much earlier. This has reduced both complications of pre-eclampsia ­ accidental haemorrhage as well as its consequential renal failure, significantly. If the obstetric team is not vigilant or if the woman is unable to get quality antenatal care, recurrent renal failure with a poor outcome can still occur. In 20% of the subjects in whom the cortical necrosis becomes predominant, renal failure is irreversible, warranting a renal transplant and dialysis. In women with persistent albuminuria in severe pre-eclampsia, there is an increased tendency for renal failure. However, the presence of albuminuria may not be indicative of an impending renal shut down. In actuality, it reflects the compromised renal function, the consequence of which is a renal shut down. Thus, proteinuria is not the cause of renal compromise and renal failure, but in actuality, it is a marker of the underlying pathology. A word about uric acid estimation for assessing renal function in pre-eclampsia will not be out of place here. Although many biochemical markers have been used to assess renal function in non-pregnant women, many of them as seen become incompetent in pregnancy more so in preeclampsia. Uric acid assessment in pre-eclampsia has been done at different times to assess renal function. One study concludes that monitoring of plasma creatinine level among patients with pre-eclampsia will help to predict those at risk of developing pre-eclampsia. In the same way, monitoring of plasma uric acid level in those with pre-eclampsia will help to predict those that will develop eclampsia. Here the role of uric acid gets reinforced as a good prognosticator rather than predictor as has been suggested in this study. It has, therefore, a limited role in reflecting the renal function in a woman with pre-eclampsia. When the level of uric acid is assessed, the state of oxidative stress in the pregnant women is being studied and not the renal function. No wonder, a subsequent study found uric acid to be unreliable for assessing renal function in pregnancy. In the former, the changes are the result of vasospasm and in the latter because of acute blood loss. This team diligently studied and documented renal changes in subjects who died in pregnancy on postmortem. They compared postmortem findings of 112 women without pre-eclampsia who died of hypertensive disorders of pregnancy with those who died of other obstetric causes. They performed postmortems within 2 hours of death, thereby increasing the accuracy of their results. Even after so many years, their results have been corroborated by images obtained through modern imaging technologies. No discussion and pathology of pre-eclampsia or eclampsia is complete without taking into account the description of these workers. The entire compilation of their work has been published in a book by Sheehan and Lynch. All this gives an advantage to workers in this field being able to study the kidneys extensively and easily. This is not to run down the involvement of kidneys in pre-eclampsia and its complications. But the ease of access to kidneys lends great importance to renal changes in pre-eclampsia. Also, there were suggestions that the aetiopathology of pre-eclampsia and its complications lies in the kidneys. It is well-known now that not the kidneys but the foetomaternal interface is at the core of the aetiopathology in pre-eclampsia. Changes emanating here involve other organs including the kidneys and systems in their processes secondarily. Until recently, science did not have an accurate and reliable means to know what is happening at the foetomaternal interface. With the advent of ultrasonography in general and colour Doppler in particular the picture changed. As a result, the accurate picture of aetiopathology became clear to the scientific investigators. We now have a reasonably precise idea of what all causes and what is the sequence of events in pre-eclampsia. Microscopic appearance can be further divided into light microscopic appearance and appearance under electron microscopic. As regards the gross appearance of kidneys in preeclampsia, there is an insignificant increase in the size of the kidneys. While one must accept that the kidneys do bear the heavy brunt of the disease of pre-eclampsia, they are not the game changers in the entire aetiopathology of pre-eclampsia. Also, changes in the renal systems can be easily studied primarily through an easily obtainable urine sample.

Syndromes

  • Allergic reaction to contrast dye
  • Increase in body fat
  • Cloudy urine
  • The cause of the drooling has not been diagnosed.
  • Pregnancy -- breast tenderness tends to be more common during the first trimester and in women who become pregnant at a young age
  • CBC with blood differential
  • Narrowing or stricture of the intestine
  • Store cans of powdered formula in a cool, dry place with a plastic lid on top. Always wash your hands and the top of the container before handling.
  • Cabbage

It was also found to exert therapeutic effect in resistant cancer cases [207 hypertension in dogs discount dipyridamole 100 mg on-line, 208] arteria femoralis communis dipyridamole 25 mg purchase otc. The cardiolipin ingredient forms stable liposomes, with effective entrapment of the drug. This formulation is currently being tested for various leukemias and cancers of breast, liver, stomach, and gonads [217]. Due to sphingomyelin contents, these formulations are also often known as sphingosomes. Encapsulation by sphingomyelins reduces their degradation in vivo-not only to prolong the t1/2, but also to reduce toxicity [218]. Both these formulations are currently being developed as therapeutic agents in advanced solid tumors. Atragen had shown a significantly increased blood t1/2 compared to uncapsulated tretinoin in clinical trials. With advancement in technology and arrival of new knowledge, liposomal formulations are getting better, with improved drug loading and more control over site-specific drug delivery. Trigger-release formulations are also giving new hope in attaining more control over the drug release. The major challenges toward fruitful translation of nanomedicines are discussed later, in Chapter 18, and these obstacles are equally valid for liposomal formulations. Watkins, Diffusion of univalent ions across the lamellae of swollen phospholipids, J. Storm, Targeted drug delivery systems for the intracellular delivery of macromolecular drugs, Drug Discov. Zhang, Repeated administration of hyaluronic acid coated liposomes with improved pharmacokinetics and reduced immune response, Mol. Gregoriadis, Dehydration-rehydration vesicles: a simple method for high yield drug entrapment in liposomes, Nat. Zhang, Thin-film hydration followed by extrusion method for liposome preparation, Methods Mol. Bialer, Prolongation of the circulation time of doxorubicin encapsulated in liposomes containing a polyethylene glycol-derivatized phospholipid - pharmacokinetic studies in rodents and dogs, Pharm. Fuks, Liposomes as in vivo carriers of adriamycin: reduced cardiac uptake and preserved antitumor activity in mice, Cancer Res. Devaux, Formation of unilamellar vesicles by repetitive freeze-thaw cycles: characterization by electron microscopy and 31P-nuclear magnetic resonance, Eur. Barenholz, Prolonged circulation time and enhanced accumulation in malignant exudates of doxorubicin encapsulated in polyethylene-glycol coated liposomes, Cancer Res. Allen, Targeted delivery and triggered release of liposomal doxorubicin enhances cytotoxicity against human B lymphoma cells, Biochim. Allen, Long-circulating (sterically stabilized) liposomes for targeted drug-delivery, Trends Pharmacol. Hua, Targeting sites of inflammation: intercellular adhesion molecule-1 as a target for novel inflammatory therapies, Front. Bendas, Immunoliposomes - a promising approach to targeting cancer therapy, BioDrugs 15(4) (2001) 215­224. Blumenthal, Lipid-based nanoparticles as pharmaceutical drug carriers: from concepts to clinic, Crit. Cullis, Liposomal drug delivery systems: from concept to clinical applications, Adv. Xin, Development of a novel liposomal nanodelivery system for bioluminescence imaging and targeted drug delivery in ErbB2-overexpressing metastatic ovarian carcinoma, Int. Yang, Drug delivery systems for differential release in combination therapy, Expert Opin. Muzykantov, Advanced drug delivery systems that target the vascular endothelium, Mol. Greish, Nanomedicine for drug targeting: strategies beyond the enhanced permeability and retention effect, Int. Maeda, A new concept for macromolecular therapeutics in cancer-chemotherapy - mechanism of tumoritropic accumulation of proteins and the antitumor agent Smancs, Cancer Res. Hope, Contact hypersensitivity: a simple model for the characterization of disease-site targeting by liposomes, Biochim. McDonald, Openings between defective endothelial cells explain tumor vessel leakiness, Am. Allen, Transendothelial movement of liposomes in vitro mediated by cancer cells, neutrophils or histamine, J. Khan, Liposomal formulations in clinical use: an updated review, Pharmaceutics 9(2) (2017) 12. Chang, Does a targeting ligand influence nanoparticle tumor localization or uptake Allen, Block copolymer micelles for delivery of cancer therapy: transport at the whole body, tissue and cellular levels, J. Drummond, Development of ligand-targeted liposomes for cancer therapy, Expert Opin. Zalipsky, In vivo fate of folate-targeted polyethylene-glycol liposomes in tumor-bearing mice, Clin.

Usage: gtt.

Laboratory diagnosis of rabies encephalopathies arteria3d viking pack dipyridamole 100 mg discount, tetanus blood pressure chart age nhs dipyridamole 25 mg cheap, listeriosis, acute disability resulting from trauma, poisoning and other viral nonsuppurative encephalitides (Dimaano, Scholand, Alera, & Belandres, 2011). Paralytic rabies in humans is often mistaken for Guillain-Barr Syndrome (Hemachudha & e Mitrabhakdi, 2000; Solomon et al. Other infections can also mask or mimic rabies leading to clinical misdiagnosis (Mallewa et al. Thus, reliable and quality-assured laboratory-based diagnostic assays are essential for confirming the presence of rabies virus or other lyssaviruses. There are currently no diagnostic tests available to detect lyssaviruses before the onset of clinical signs. However, there are a number of highly reliable, sensitive, and specific assays that have now been established globally in quality-assured diagnostic laboratories. Such tests are pivotal for ensuring rapid case management with respect to human and animal rabies exposures, including the implementation of lifesaving public and animal health control, including effective pre- and postexposure prophylaxis (Ma et al. Rapid positive diagnosis supports the case for an urgent public health investigation and medical care for all clinical and potentially exposed persons, as well as examination and rabies booster vaccination of currently vaccinated pets and livestock. It also allows clinicians to cease the investigations or interventions initiated to cover other disorders in the differential diagnosis. A negative diagnosis can be as important as a positive diagnosis by ensuring other conditions are immediately considered in clinical cases and ruling out the need for rabies control prophylaxis for both human and animal exposures. In the absence of diagnostic testing, public and animal health control, involving significant resources, may need to be applied in every suspect case to minimize the risk of rabies deaths. In this regard, the indication for rabies testing is directly related to an exposed human or domestic animal. This is often referred to as passive surveillance in that the submitted animals are selected on the basis of observed clinical signs and their encounters with humans and animals. In the United States in 2017, 93,651 samples were screened for rabies of which 4454 (4. The majority of the reported cases in Europe were detected in domestic animals (dogs, cats, and cattle accounted for $60%) and foxes (29%). Bats (42 cases) accounted for only 1% of the reported rabies cases in Europe in 2017. The distribution and epidemiology of rabies cases will thus vary by area and to a certain degree will be reliant upon and reflect sampling bias. Some countries have never initiated wildlife or bat surveillance programs, while in other regions. Test results on the submitted animals in passive surveillance schemes are thus not representative of population-based testing in the various species. Whereas active surveillance is necessary to gain good epizootiological knowledge on rabies, limited information can be enhanced through careful specimen acceptance policies. An optimal understanding of local and regional transmission patterns will help guide risk assessment for potentially exposed humans and animals, especially in situations where the exposing animal is not available for diagnostic testing or observation (Anonymous, 2011). In addition to the testing of animals that commonly contribute to disease transmission, rabies surveillance can be enhanced by the acceptance of uncommonly tested animals with neurological signs compatible with rabies, such as deer, bear, beaver, badger, or other potential spill over species. For heightened surveillance, vigilance and typing of viral strains (or lyssavirus species) may identify new and emerging viruses and hosts. This is particularly important in areas where rabies control efforts are ongoing through methods, such as oral vaccination or trap-vaccinate-and-release. In passive or active bat surveillance programs, the speciation of the individuals is extremely important because the risk of rabies may vary according to bat species, and the viral variants that are found in these animals are diverse. When rabies is confirmed, the diagnosis allows initiation of human postexposure prophylaxis for potentially exposed persons and the management of potentially exposed domestic animals. The difficulty with human and domestic animal encounters with these animals is that bats are comparatively small bodied. A potential exposure, such as a bite, from these animals may be ignored by humans because the actual trauma from a bite may be painless or unremarkable. In some countries, such as the United Kingdom, clinicians offer precautionary post exposure prophylaxis following any bite from a bat. However, in other countries, some humans may not have awoken from slumber when they were bitten by a bat, or may have been unable to report a bite if noticed. It is important to understand the risk of rabies following bites from pet animals in different countries and whether euthanasia and testing is demanded for reasons other than a genuine suspicion of disease. Observation or quarantine may be considered more appropriate than euthanasia and testing (Anonymous, 2011). Laboratory diagnosis of rabies However, in some developed countries where wildlife rabies is present, if a concern about a potential exposure is voiced to health authorities and the pet is euthanized immediately after the biting incident, it is often tested for rabies in the absence of clinical signs. Thus, some diagnostic laboratories may test numerous healthy but unwanted domestic dogs and cats. In countries free of terrestrial rabies, the travel movements and compliance of the pet are also important when determining risk and need for rabies testing. During sample preparation and testing, the most likely risk of exposure is through accidental penetrating injuries with contaminated laboratory equipment or exposure of mucous membranes or broken skin to infectious tissue or fluids. The highest viral concentrations are found in central nervous system tissue, salivary glands, and saliva, but any innervated tissue may be a source of virus exposure. The level of protection required by law will differ globally and likely reflects both the biological properties of the virus and the disease status in the country. Some countries may also control the handling of rabies virus and infected material under antiterrorism and national security regulations. In the United Kingdom, the highest biosafety level for animal pathogens is assigned to rabies virus and specific licenses are required to handle live virus within highly expensive and regulated high containment laboratories.

References

  • Liu T, Soong S. Epidemiology of malignant melanoma. Surg Clin North Am 1965;76:1205-1222.
  • Woodcock TE, Murkin JM, Farrar JK, et al: Pharmacologic EEG suppression during cardiopulmonary bypass: Cerebral hemodynamic and metabolic effects of thiopental or isoflurane during hypothermia and normothermia, Anesthesiology 67:218, 1987.
  • Pak CY, Fuller CJ: Assessment of cystine solubility in urine and of heterogeneous nucleation, J Urol 129(5):1066n1070, 1983.
  • Moreau D. Physician's drug handbook. Springhouse: Springhouse Corporation; 1995.
  • Greenberg A, Verbalis JG. Vasopressin receptor antagonists. Kidney Int. 2006;69(12):2124-2130.