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Remember antibiotics for sinus infection treatment ampicillin 500 mg on line, though antibiotics z pack and alcohol buy ampicillin 250 mg free shipping, these patients often have a normal cardiac output at the time of diagnosis-because of the worsening ventricular failure! Emergency Treatment for Severe Heart Failure [Know:] With severe ventricular failure, patients may require short-term treatment with inotropes (dopamine, dobutamine, and milrinone). Dobutamine is another inotropic agent that can be used for severe ventricular failure. It does not have the vasoconstrictor activity of dopamine and actually has some vasodilatory effects. Both Dressler syndrome and postpericardiotomy syndrome are autoimmune processes that occur several weeks after the precipitating event. Patients with pericarditis commonly present with very severe chest pain, sometimes pleuritic, which (classically) improves when leaning forward. The pain is retro-sternal and left precordial, and referred to the neck, arms, or left shoulder. Do not treat idiopathic pericarditis with steroids because there can be a relapse when they are stopped. Constrictive pericarditis must be differentiated from restrictive cardiomyopathy (page the signs and symptoms can be similar. Pericardiectomy often does not have good results and is tried only after medical treatment options have been exhausted. On the other hand, pericardiocentesis rarely helps in diagnosis but is often used to treat viral, idiopathic, neoplastic, hypothyroid, and renal failure-related tam ponade. A lateral chest x-ray that shows calcification over the right ventricle is pathognomic for constrictive pericarditis. The pericardium can be of normal thickness in -20-25% of cases of constrictive pericarditis. In both tamponade and constrictive pericarditis, cardiac cath shows the same pressure during diastole in all 4 chambers. You can often make the differentia tion between tamponade and constrictive pericarditis at the bedside using these hallmark signs (see tamponade below). Constrictive pericarditis must be treated with an open thoracotomy and pericardiectomy. Compare and know the difference between this and constrictive pericarditis (above). If there is a> 2:1 left-to-right (pulmonary/systemic) shunt, a surgical closure is done, even if the patient is asymp · What are the 2 clinical hallmarks of constrictive pericarditis Patients with ostium primum atrial septal defect may have a loud pansystolic murmur 2° to mitral and/or tricuspid regurgitation. Know that the magnitude of any shunt does not depend on the total blood flow rate, but is commonlyly a constant ratio of pulmonic to systemic flow (Qp/Qs). It is the most common form of congenital heart disease found initially in adults (F> M), excluding a bicuspid aortic valve. The left-to-right shunt causes diastolic overloading of the right ventricle and increased pulmonary blood flow with inspiration and expiration. They are uncommon in adults because most have either closed spontaneously or have been surgically closed in childhood. Other associated anomalies include mitral valve problems, left ventricular myocardium problems, and membranes in the left atrium. Notice that all of the heart problems associated with coarctation of the aorta are left-sided! The classic physical findings are either a delayed fem oral/brachial pulse (feeling the brachial and femoral pulses, there is a distinct delay in femoral pulse) or an absent femoral pulse. Patients can have upper-body hypertension and can get hypertensive aneurysmal dilatation and rupture of the circle of Willis. Look for rib notching on chest x-ray due to the collateral vessels getting very large and eroding the ribs. Turner syndrome is associated with coarctation of the aorta and a bicuspid aortic valve. These two are covered extensively in Pulmonary Medicine, Book 2, so we will cover the other causes here. This can present as exertional chest pain or exertional syncope in a young, otherwise healthy individual. Syncope after exercise can occur in "normal" people, but syncope during exercise is never normal. With anomalous coronary artery, there is an abnormal course of I of the 2 coronary arteries between the 2 great vessels, the pulmonary artery and aorta. At rest, there is plenty of room for the vessel to pass without compromise; however, in extreme exercise, the cardiac output can increase 4-8-fold. This expands the elastic pulmonary artery and aorta, resulting in compression of the coronary artery as it courses between the great vessels. Most patients treated for acute pulmonary thromboembolism do not develop chronic pulmonary hypertension. Next most common are coronary anomalies (17%)-although this is a more likely cause in the 30-40-year-old group. It is absolutely contraindicated in the I st trimester; although, to be safe, most physicians do not give it at all during pregnancy. Although heparin is not contraindicated, it does cause increased morbidity and mortality in mother and child. A maternal a rubella infection during pregnancy is common cause among of supravalvular aortic is stenosis, not a · What is the only effective treatment for Eisenmenger syndrome Previously it was called idiopathic pulmonary has 1: alone, pregnancy is associated with increased risk; refer these patients to a high-risk obstetrician.

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Infection in organ transplantation: risk factors and evolving patterns of infection antibiotic resistance reversal buy ampicillin 500 mg with mastercard. Prophylactic antibiotics are justified for patients with immunodeficiencies antibiotics for staph safe 500 mg ampicillin, severe manifestations, or history of cardiac, kidney, or inflammatory bowel disease. Antibiotic management of febrile neutropenia: current developments and future directions. Antimicrobial prophylaxis and outpatient management of fever and neutropenia in adults treated for malignancy: American Society of Clinical Oncology clinical practice guideline. Clinical practice guideline for the use of antimicrobial agents in neutropenic patients with cancer: 2010 update by the lnfectious·Diseases Society of America. Loperamide can be used cautiously with the antibiotics but should not be used alone as a form of treatment in severe disease. Mechanisms of action and clinical appli cation of macrolides as immunomodulatory medications. Declining susceptibilities of gram negative bacteria to the fluoroquinolones: effects on pharma cokinetics, pharmacodynamics, and clinical outcomes. Newer beta-lactam antibiotics: doripenem, ceftobiprole, ceftaroline, and cefepime. Therapeutic monitoring of vancomycin in adult patients: a consensus review of the American Society of Health-System Pharmacists, the Infec tious Diseases Society of America, and the Society of Infec tious Diseases Pharmacists. Combination antibiotic therapy for empiric and definitive treatment of gram-negative infections: insights from the Society of Infectious Diseases Pharmacists. Appraising contemporary strategies to combat multidrug resistant gram-negative bacterial infections-proceedings and data from the Gram-Negative Resistance Summit. Guidelines for diagnosis, treatment, and prevention of C/os lridi11111 difficile infections, 2013. Clinical practice guidelines by the Infectious Diseases Society orAmerica for the treatment or mcthicillin-rcsistant S1aphrloc ocrn. Clinical practice guideline for the diagnosis and management of group A streptococcal pharyngitis: 2012 update by the Infectious Diseases Society or America. Antifungal drug resistance: mechanisms, epide miology, and consequences for treatment. Emergence, control and re-emerging leptospirosis: dynamics of infection in the changing world. Clinical practice guidelines for the management or cryptococcal diseasc: 20 I 0 update by the infectious diseases society of America. Clinical practice guidelines for the management of patients with histoplasmosis: 2007 update by the Jnlectious Diseases Society of America. Hantavirus infections for the clinician: from case presentation to diagnosis and treatment. Therapeutic strategies for the prevention and treatment of cytomegalovirus infection. Seasonal influenza in adults and children-diagnosis, treatment, chcmop ro phylaxis, and institu tional outbreak management: clinical practice guidelines of the lnlectious Diseases Society of America. International consensus guide lines on the management or cytomcgalo v irus in solid organ al; Center for Disease Control and fl! Come fly with me: review of clinically important arboviruses for global travelers. Progress and problems in understanding and managing primary Epstein-Barr virus infec tions. Clinical features of viral meningitis in adults: significant differences in cerebrospinal fluid findings among herpes simplex virus, varicella zoster virus, and enterovirus infections. Shigellosis update: advancing antibi otic resistance, investment empowered vaccine development, and green bananas. Update on tuberculosis of the central nervous system: pathogenesis, diagnosis, and treat ment. Campylobac/er jejuni: a brief over view on pathogenicity-associated factors and disease-mediat ing mechanisms. Surgical management of endocardi tis: the society of thoracic surgeons clinical practice guideline. Society for Healthcare Epide miology of America; I nfectious Diseases Society of America. Infectious Diseases Society of America; European Society for Microbiology and Infectious Diseases. International clinical practice guidelines for the treatment of acute uncomplicated cystitis and pyelonephritis in women: A 20 I 0 update by the Infectious Diseases Society of America and the European Society for Microbiology and Intectious Diseases. Practice parameter: treatment of nervous system Lyme disease (an evidence-based review): report of the Quality Standards Subcommittee of the American Academy of Neurology. Diagnosis, prevention, and treatment of catheter-associated urinary tract infection in adults: 2009 International Clinical Practice Guidelines from the Infectious Diseases Society or America. Clinical practice guidelines by the infectious diseases society of America for the treatment of methicillin-resistant Staphy lococcus aureus infections in adults and children. Guideline for the prevention and control of norovirus gastroenteritis outbreaks in healthcare settings. Asthma and Immunology; American College of Allergy, Asthma and Immunology; Joint Council of Allergy, Asthma and Immunology. Practice guidelines for the diagnosis and manage ment of skin and soft-tissue inlcctions. Prevention of infective cndo carditis: guidelines from the American Heart Association: a guideline from the American Heart Association Rheumatic Fe ver, Endocarditis and Kawasaki Disease Committee.

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Clinical features are intermediate and variable and can include moderately severe hemolytic anemia but often with avoidance of transfusions until adulthood virus 2 game cheap ampicillin online. There are 3 categories of -thalassemia: -thalassemia minor (heterozygotes): mild or no anemia infection control nurse 250 mg ampicillin purchase amex, with a disproportionately high number of microcytes. In most patients, this disorder has 2- to 3-fold elevations of HbA2 (a2o2) and slight increases in HbF (a2y2) on hemoglobin electrophoresis. The remaining, highly insoluble a-globin precipitates into homotetramers, or inclusion bodies, which are toxic to erythrocytes and cause them to die within the marrow. Surviving erythrocytes carry inclusion bodies that are detected by the spleen, leading to removal of the erythrocytes and chronic hemolytic anemia. The resulting severe anemia (developing over the I 51 year of life) results in elevated erythropoietin levels and thus, erythroid hyperplasia. If the erythroid hyperplasia is severe, it can lead to extramedullary hematopoiesis in the liver and spleen and an expanded bone marrow with the latter, giving children "chipmunk facies. Think about a megaloblastic process in patients who present with a macrocytic anemia, pancytopenia, and slight indirect hyperbilirubinemia (from the continuous low-level intramedullary hemolysis). In addition to anemia, deficiencies in B12 also produce gastrointestinal effects (smooth sore tongue, diarrhea) and neurological deficits (ranging from paresthesias to frank psychosis). B12 deficiency may be present without 3) -thalassemia intennedia (homozygous): Not all patients with homozygous defects of -globin production have the full clinical severity described above. The term "-thalassemia intermedia" is used to convey this hetero geneity and to describe those patients who range from the asymptomatic to the transfusion-dependent states. If you highly and extravascular hemolysis, released hemoglobin is quickly bound to haptoglobin and then engulfed by macrophages. The resultant low level of haptoglobin can be used to diagnose hemolysis-but does not help distinguish the type. The Schilling test, in which the fate of radiolabeled B12 ingested by the patient is fol lowed, was previously used to confirm the diagnosis but is rarely used and generally not available anymore. Treat B12 deficiency with daily injections for folate deficiency with daily oral replacement. Heme loses of the iron and is converted more of the to bilirubin and cleared in the urine or stool. Urine hemosiderin high = 1 week, then weekly injections for 1 month, then monthly. Common oxidative stressors are infections, medications (including dapsone, sulfa drugs, and antimalarials), fava beans, and diabetic ketoacidosis. Other hematologic findings (and buzzwords) include Heinz bodies (chunks of dena tured hemoglobin) on special smears and "bite cells" in peripheral blood. Patients are asymptomatic or demonstrate renal papillary necrosis, painless hematuria, and isosthenuria. Splenic sequestration is common and can occur both in children and adults (unlike in sickle cell anemia), because the spleen does not always undergo early autoinfarction. Pyruvate kinase deficiency and other enzyme deficiencies within the glycolytic pathway are subject to hemolytic crisis without exposure to oxidative stress. You can screen prospective parents for the carrier state and provide genetic counseling. Exchange transfusion is sometimes required for treatment of priapism, cerebral sickling, aplastic crisis, and acute chest syndrome. Occasionally, more conservative ther apy with a simple transfusion and/or supportive care treats these presentations. A partial exchange transfusion program is reserved for those with a history of stroke. Medications such as hydroxyurea are used to increase HbF production, which offers some protection against sickle crisis. Hereditary Spherocytosis and Elliptocytosis Sickle cell syndromes result from a mutation in the Val) -globulin gene (6Giu, in which valine is substi tuted in place of glutamine. Know that parvovirus 8 19 may cause either a pure red cell aplasia or a worsening of anemia by decreasing erythropoiesis in the face of chronic hemolysis. Recurrent tissue infarction, microinfarcts of and the these are autosomal dominant disorders of the R8C cytoskeleton that result in loss of membrane flexibility and are associated with chronic hemolysis. These disorders of spherocytosis and elliptocytosis are seen in Northern European populations. Complications may include cholelithiasis, due to bilirubin stones, and splenomegaly. Think about these disorders in patients who demonstrate evidence of hemolysis and have spherocytes or elliptocytes on their peripheral smear. Recurrent infarcts of the spleen lead to functional asplenia with increased risk of infection from encapsu Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus injluenzae, and from Salmonella. To protect against these organisms, penicillin prophylaxis is used in children until age 5. Sophisticated molecular membrane studies (usually available at research institutions) can be done to make a definitive diagnosis. Acute chest syndrome (chest pain and desaturation of oxygen) is thought to represent microinfarctions of the lung. The acute chest syndrome is a hematologic emergency which may require red cell exchange transfusion.

Syndromes

  • Limit alcohol to one drink per day (women who are at high risk for breast cancer should consider not drinking alcohol at all).
  • Breast cancer
  • Family troubles - resources
  • Feeling the joint and muscles for tenderness
  • Diagnose the cause of blood in stools, diarrhea, or very hard stools (constipation)
  • Activated charcoal
  • Jaundice (yellow coloring of the skin or eyes)
  • Wear a medical alert tag if you have a pre-existing breathing condition, such as asthma.
  • Reduced urine output

In the past virus hoax generic ampicillin 250 mg buy on line, mercurial diuretics were commonly associated with the development of Fanconi syndrome antibiotic knee spacer surgery ampicillin 250 mg with mastercard. The afflicted children typically present with extremely severe metabolic acidosis, growth retardation, nephrocalcinosis, and nephrolithiasis. Hypokalemia, which is usually present, may actually be caused by the associated sodium depletion and stimulation of the reninangiotensin-aldosterone axis. Therefore, renal potassium losses decrease considerably when appropriate therapy with sodium bicarbonate is instituted. These include (a) a defect in sodium reabsorption where a favorable transepithelial voltage cannot be generated and/or maintained, and (b) hypoaldosteronism. Other causes include cyclosporin nephrotoxicity, renal allograft rejection, sickle cell nephropathy, and many autoimmune disorders, such as lupus nephritis and Sjögren syndrome. In contrast to hypoaldosteronism, urinary acidification is impaired in these subjects. Other patients with hyporeninemic hypoaldosteronism have a more complex pathophysiology. This is occasionally seen with trauma resuscitation or during treatment of right ventricular myocardial infarction. As a result, patients taking topiramate have a 2- to 4-fold increased incidence of calcium phosphate kidney stones, which is discussed further in Chapter 13. In fact, this is actually quite common if alkali-generating compounds (eg, acetate or lactate) are not administered concomitantly with amino acids; however, replacement of the chloride salt of these amino acids with an acetate salt easily avoids this problem. It turns out that it is metabolism of sulfur-containing amino acids that obligates excretion of acid because neutrally charged sulfur is excreted as sulfate. In general, 1 g of amino acid mixture generally requires 1 mEq of acid to be excreted. Ergo, the acetate content of parenteral alimentation should probably match the amino acid content on a mEq/g basis. Potassium-Sparing Diuretics Aldosterone antagonists (eg, spironolactone and eplerenone) or sodium channel blockers (eg, amiloride and triamterene) may also produce a hyperchloremic acidosis in concert with hyperkalemia. Trimethoprim and pentamidine may also function as sodium channel blocker and cause hyperkalemia and hyperchloremic metabolic acidosis. The diagnosis of lactic acidosis must be considered in all forms of metabolic acidosis associated with an increased anion gap, particularly those cases associated with local or systemic decreases in oxygen delivery, impairments in oxidative metabolism, or impaired hepatic clearance. Diabetic ketoacidosis results from lack of sufficient insulin necessary to metabolize glucose and excess glucagon that causes the generation of short-chain fatty ketoacids. Ethylene glycol and methanol ingestion are important causes of an anion gap metabolic acidosis that are associated with an elevated osmolar gap. Metabolic acidosis in the setting of acute and chronic renal failure is generally not severe. In other words, the increase in the anion gap roughly parallels the fall in bicarbonate concentration; however, during therapy, renal perfusion is often improved and substantial loss of ketoanions in urine may result. The fundamental principles of acid­base therapy are that a diagnosis must be made and treatment of the underlying disease state initiated. With most of the hyperchloremic states of metabolic acidosis, gradual correction of the acidosis is effective and beneficial. Oral bicarbonate or an anion whose metabolism results in bicarbonate generation is generally preferred. The acute treatment of metabolic acidosis associated with an increased anion gap with intravenous sodium bicarbonate is controversial. Unfortunately, there is little in the way of randomized clinical data to guide us. Based primarily on experimental models, it appears that bicarbonate therapy may actually be deleterious in this setting, especially if the acidosis is associated with impaired tissue perfusion. The so-called paradoxical intracellular acidosis that results when bicarbonate is infused during metabolic acidosis probably accounts for a portion of these deleterious effects. Experimentally, administration of sodium bicarbonate in models of metabolic acidosis is associated with a fall in pHi in several organs, including heart. The hypertonic state itself may impair cardiac function, especially in patients undergoing resuscitation for cardiac arrest. Based on these data, we do not support therapy with intravenous sodium bicarbonate for acute anion gap metabolic acidosis in the emergency situation. Mechanism of "paradoxical" intracellular acidosis following administration of sodium bicarbonate. Dichloroacetate, which is specifically designed to decrease lactate production in lactic acidosis, was used in animals with some success. Perhaps more concerning is that none of these agents are still protected by patents, and it is unclear who (if anyone) will bear the cost of studies necessary to demonstrate their clinical safety and efficacy. Mechanisms of the effects of acidosis and hypokalemia on renal ammonia metabolism. Hyperchloremic metabolic acidosis is usually effectively treated by gradual correction of acidosis with administration of bicarbonate. Acute treatment of an anion gap metabolic acidosis with intravenous sodium bicarbonate may be deleterious, especially in conditions associated with impaired tissue perfusion. The administration of sodium bicarbonate in animals with metabolic acidosis is associated with a fall in pHi in several organs, as well as additional hemodynamic compromise. When H+ losses exceed the daily H+ load produced by metabolism and diet, a net negative H+ balance results. As a result serum 3 bicarbonate will not rise unless there is a change in renal bicarbonate handling (maintenance factor).

Usage: p.c.

Hypothyroidism antibiotic resistance nature 250 mg ampicillin order overnight delivery, diabetes antibiotic resistance experts 250 mg ampicillin buy fast delivery, are also associated amyloidosis Ganglion Cyst Ganglion cysts can occur at any joint or tendon sheath, but they are most commonly found on the dorsum of the wrist at the scapholunate joint. There is no communication between the inside of the joint capsule and the interior of the ganglion. They are usually asymptomatic but may cause pain due to compression of a nerve or joint space. Ganglions generally are not treated, but temporary resolution may be provided by firm pressure or aspiration. But this should be avoided because it may cause an inflammatory response and recur. It is "unstuck" only with strong effort or with passive movement using the other hand-which causes significant pain. There is tenderness at the base of the finger (palmar aspect); often a tendon nodule can be felt. The cause is swelling of the flexor tendon and the opening of the flexor tendon sheath at the base of the finger. Splinting and local steroid injections can help, but a simple surgery is required to cure the condition. De Quervain Tenosynovitis this is a chronic or subacute inflammation of the flexor tendons or the abductor pollicis longus tendon of the thumb. It is characterized by pain and well-localized tenderness over the styloid process of the distal radius. It is often caused by repetitive twisting of the wrist with certain motions, like wringing clothes. The Finkelstein test (forced ulnar motion of the wrist with the thumb Hip Trochanteric Bursitis this bursitis is the most common cause of lateral thigh discomfort. Patients report "hip" pain when lying on the involved side, draping the involved leg over the non-involved limb, or bearing weight on the affected © 2014 MedStudy-Piease Report Copyright Infringements to copyright@medstudy. Patients with femoral neck fractures or traumatic hip dislocations are especially susceptible because the blood supply to the femoral head is disrupted. This helps distinguish bursa pain from true hip joint pain, which causes a point of maximum intensity in the groin (may radiate to the buttock). The compromised blood supply can be due to trauma, certain medical conditions, medications/drugs, or idiopathic disease. It most commonly affects the epiphysis (ends) of the femur (affecting the hip > the knee. Patients may complain of morning stiffness occurs after inactivity and resolves with use). Standing or "weight-bearing" radiographs show joint-space narrowing +/- subchondral sclerosis and/or osteophytes. In patients with typical pain and abnormal radiographs, no further imaging is necessary. This forms a synovial, fluid-filled sac in the midline behind the knee or in the upper calf. A Baker cyst usually occurs as a result of chronic arthritic conditions in which there is persistent synovial effusion. If an arthritic patient with knee involvement presents with a painful swollen calf, suspect a ruptured Baker cyst causing pseudo-phlebitis. On exam, the cyst can be palpated in the posterior knee when the knee is partially flexed. If the cyst is very large or causes significant pain, you can aspirate the knee (not the back of the knee! Conservative measures should be exhausted (especially weight loss, physical therapy, and use of assist devices, such as canes) before referral for total hip arthroplasty. Pes Anserine Bursitis this benign neuroma causes painful, burning paresthesias and tenderness in the interdigital webbing due to repeated nerve trauma. It is much more common in women and thought to be related to high heels and tight fitting shoes. Treatment includes lowering the heel and wearing wider, soft-soled shoes with metatarsal arch support. Glucocorticoid injections may be helpful, and severe cases may require surgical excision of the nerve. This is just proximal to the area where the 3 tendinous extensions of the gracilis, sartorius, and semi-tendinous muscles insert into the medial aspect of the tibial tuberosity. Radiographs are usually not necessary, but can assist in excluding diseases that present similarly, such as idiopathic, monoarticular, benign synovial tumor that causes recurrent hemarthrosis, usually of the knee in young adults. Patients have recurrent bleeding into the knee, resulting in a darkly pigmented joint aspirate. There is no known etiology, although it seems to be associated with osteopenia and osteoarthritis. Weight-bearing pain is present initially on the medial aspect of the knee, and symptoms often resolve with conservative management, including protected weight bearing and analgesics. Look for evidence of a spondyloarthropathy in any patient who presents with plantar fasciitis. Recurrent steroid injections should be avoided because they cause fat pad atrophy. By imaging, you are ruling out: ·Disk herniation · Spinal stenosis · Compression fracture ·Malignancy ·Infection Muscle Strain More than 50% of adults experience at least I episode of back strain at some time. Classic presentation is agonizing, lower back pain with a history of lifting a heavy object or making a sudden movement. Physical exam typically reveals guarding of movement due to pain and no true muscle weakness or neurologic deficit. These include a moderately prolapsed disk, catching of the synovial membrane in a facet joint, transient subluxation with ligament strain, and basic muscle strain.

References

  • Jones D, Renshaw AA. Recurrent crystal-storing histiocytosis of the lung in a patient without a clonal lymphoproliferative disorder. Arch Pathol Lab Med 1996;120:978-80.
  • Jensen RT, Berna MJ, Bingham DB, et al: Inherited pancreatic endocrine tumor syndromes: Advances in molecular pathogenesis, diagnosis, management, and controversies. Cancer 113:1807, 2008.
  • Rainier S, Johnson LA, Dobry CJ, et al. Relaxation of imprinted genes in human cancer. Nature 1993;362(6422):747-749.
  • Craggs JG, Price DD, Verne GN, et al. Functional brain interactions that serve cognitive-affective processing during pain and placebo analgesia. Neuroimage. 2007;38(4):720-729.
  • Belicki K, Cuddy M. Nightmares: facts, fictions and future directions. In Gackenbach J, Sheikh A, eds. Dream Images: A Call to Mental Arms. Amityville, NY: Baywood Publishing Co.; 1991: pp. 99-113.
  • Goodstadt L, Ponting CP. Sequence variation and disease in the wake of the draft human genome. Hum Mol Genet 2001;10:2209-14.
  • Bugiani O, Giaccone G, Rossi G, et al. Hereditary cerebral hemorrhage with amyloidosis associated with the E693K mutation of APP. Arch Neurol 2010;67(8):987-95.