Aciphex

Aciphex 20mg

• 30 pills - $27.54
• 60 pills - $45.17
• 90 pills - $62.80
• 120 pills - $80.42
• 180 pills - $115.68
• 270 pills - $168.56
• 360 pills - $221.44

Aciphex 10mg

• 60 pills - $41.08
• 90 pills - $53.24
• 120 pills - $65.40
• 180 pills - $89.72
• 270 pills - $126.20
• 360 pills - $162.68

Currently chronic gastritis journal 10 mg aciphex for sale, asthma medications are classified according to their time of action in the overall management of this disease gastritis symptoms in child discount aciphex 10 mg without a prescription. They are either quick-relief medications, such as -adrenergic agonist bronchodilators to reverse smooth-muscle constriction, or long-term control medications, such as inhaled corticosteroids, long-acting -agonist bronchodilators, and leukotriene modifiers. This section of the lung from a patient with asthma shows a bronchiole in the center with surrounding alveoli. This high-magnification photomicrograph shows the structure of a bronchiolar pseudostratified columnar epithelium containing a large number of goblet cells. Note the presence of a large number of eosinophils (cells with red cytoplasm), lymphocytes, and other connective tissue cells that infiltrated the lamina propria and submucosa of the bronchiole. The smooth muscle layer is also thick and the underlying adventitia contains enlarged blood vessels. Occasional brush cells and densecore granule cells are also present along the length of the respiratory bronchiole. Alveoli are the sites at which air leaves and enters the bronchiole to allow gas exchange. Alveoli are the terminal air spaces of the respiratory system and are the actual sites of gas exchange between the air and the blood. Each alveolus is surrounded by a network of capillaries that brings blood into close proximity to inhaled air inside the alveolus. The alveoli are surrounded and separated from one another by a thin connective tissue layer, the interalveolar septa, containing blood capillaries. On the right is the lung surface, which is covered by visceral pleura containing simple squamous epithelium and an underlying layer of connective tissue. The junctions form an effective barrier between the air space and the components of the septal wall. These cuboidal cells are interspersed among the type I cells but tend to congregate at septal junctions. They are rich in a mixture of phospholipids, neutral lipids, and proteins that is secreted by exocytosis to form an alveolar-lining, surface-active agent called surfactant. After lung injury, they proliferate and restore both types of alveolar cells within the alveolus. Surfactant decreases the alveolar surface tension and actively participates in the clearance of foreign materials. Rings of smooth muscle are present in the knob-like interalveolar septa (see the next paragraph). Alveolar sacs usually occur at the termination of an alveolar duct but may occur anywhere along its length. Alveoli are surrounded and separated from one another by an exceedingly thin connective tissue layer that contains blood capillaries. Surfactant synthesis in the fetus occurs after the 35th week of gestation and is modulated by a variety of hormones, including cortisol, insulin, prolactin, and thyroxine. Without adequate secretion of surfactant, the alveoli would collapse on each successive exhalation. In addition, administration of cortisol to mothers with threatened premature delivery decreases neonatal mortality. Surfactant proteins help organize the surfactant layer and modulate alveolar immune responses. The alveolar surface forms a vulnerable biologic interface that is subject to many destabilizing surface forces and to continuous exposure to inhaled particles, pathogens, and toxins. The alveolar epithelium is composed of several specialized cells and their products, some of which play defensive and protective roles: In addition to phospholipids, hydrophobic proteins are necessary for the structure and function of surfactant. This electron micrograph shows two alveolar spaces separated by an alveolar septum containing capillaries, some of which contain red blood cells. Photomicrograph of an alveolus for comparison with the alveolar wall as seen in an electron micrograph. Connective tissue cells and fibers that may be present between the two basal laminae widen the air­blood barrier. It is thought that most gas exchange occurs across the thin portion of the barrier. The thick portion is thought to be a site in which tissue fluid can accumulate and even cross into the alveolus. Lymphatic vessels in the connective tissue of the terminal bronchioles drain fluid that accumulates in the thick portion of the septum. Alveolar macrophages remove inhaled particulate matter from the air spaces and red blood cells from the septum. Alveolar macrophages are unusual in that they func- the alveolar septum is the site of the air­blood barrier. The air­blood barrier refers to the cells and cell products across which gases must diffuse between the alveolar and capillary compartments. Alveolar macrophages are derived from blood monocytes and belong to the mononuclear phagocyte system (see page 181). Some engorged macrophages pass up the bronchial tree in the mucus and are disposed of by swallowing or expectoration when they reach the pharynx. Thus, at autopsy, the lungs of urban dwellers and smokers usually show many alveolar and septal macrophages filled with carbon particles, anthracotic pigment, and birefringent needle-like particles of silica. Alveolar macrophages also phagocytose infectious organisms such as Mycobacterium tuberculosis, which can be recognized in the cells in appropriately stained specimens. These bacilli are not digested by macrophages; however, other infections or conditions that damage alveolar macrophages can cause release of the bacteria and recurrent tuberculosis.

Aciphex dosages: 20 mg, 10 mg
Aciphex packs: 30 pills, 60 pills, 90 pills, 120 pills, 180 pills, 270 pills, 360 pills

Cell adhesion molecules play important roles in cell-to-cell and cell-to-extracellular matrix adhesions chronic gastritis what not to eat buy aciphex pills in toronto. The easiest way for many viruses diet by gastritis discount aciphex 10 mg free shipping, bacteria, and parasites to successfully compromise the protective functions of the epithelial layer is to destroy the junctional complexes between epithelial cells. Several proteins found in junctional specializations of the cell membrane are affected by molecules produced or expressed by these pathogenic agents. The oncogenic effect of these interactions is attributed, in part, to the sequestration and degradation of the zonula occludens and the tumor-suppressor proteins associated with the viruses. Bacteria A common bacterium that causes food poisoning, Clostridium perfringens, attacks the zonula occludens junction. This microorganism is widely distributed in the external environment and is found within the intestinal flora of humans and many domestic animals. Food poisoning symptoms are characterized by intense abdominal pain and diarrhea that begins 8 to 22 hours after eating foods contaminated by these bacteria. Binding to claudins prevents their incorporation into the zonula occludens strands and leads to malfunction and breakdown of the junction. Dehydration that occurs with this type of food poisoning is a result of a massive movement of fluids via paracellular pathways into the lumen of the intestines. Helicobacter pylori, another bacterium, resides within the stomach and binds to the extracellular domains of zonula occludens proteins. The loss of the protective epithelial barrier in the lung exposes the lung to inhaled allergens and initiates an immune response that can lead to severe asthma attacks. For simplicity of this drawing, the associated intracellular attachment proteins are not shown. The cytoplasmic domains are linked through a variety of intracellular proteins to components of the cell cytoskeleton. Through this interaction, cadherins convey signals that regulate mechanisms of growth and cell differentiation. Cadherins control cell-to-cell interactions and participate in cell recognition and embryonic cell migration. E-cadherin, the most studied member of this family, maintains the zonula adherens junction between epithelial cells. Integrins are represented by two transmembrane glycoprotein subunits consisting of 15 and 9 chains. This composition allows for the formation of different combinations of integrin molecules that are able to interact with various proteins (heterotypic interactions). Integrins interact with extracellular matrix molecules (such as collagens, laminin, and fibronectin) and with actin and intermediate filaments of the cell cytoskeleton. Through these interactions, integrins regulate cell adhesion, control cell movement and shape, and participate in cell growth and differentiation. Selectins are expressed on white blood cells (leukocytes) and endothelial cells and mediate neutrophil­ endothelial cell recognition. This heterotypic binding initiates neutrophil migration through the endothelium of blood vessels into the extracellular matrix. Selectins are also involved in directing lymphocytes into accumulations of lymphatic tissue (homing procedure). Many molecules involved in immune reactions share a common precursor element in their structure. However, several other molecules with no known immunologic function also share this same repeat element. Together, the genes encoding these related molecules have been defined as the immunoglobulin gene superfamily. It is one of the largest gene families in the human genome, and its glycoproteins perform a wide variety of important biologic functions. These proteins play key roles in cell adhesion and differentiation, cancer and tumor metastasis, angiogenesis (new vessel formation), inflammation, immune responses, and microbial attachment, as well as many other functions. At these sites, cadherins maintain homotypic interactions with similar proteins from the neighboring cell. They are associated with a group of intracellular proteins (catenins) the integrity of epithelial surfaces depends in large part on the lateral adhesion of the cells with one another and their ability to resist separation. Although the zonula occludens involves a fusion of adjoining cell membranes, their resistance to mechanical stress is limited. Reinforcement of this region depends on a strong bonding site below the zonula occludens. Like the zonula occludens, this lateral adhesion device occurs in a continuous band or belt-like configuration around the cell; thus, the adhering junction is referred to as a zonula adherens. The zonula adherens is composed of the transmembrane cell adhesion molecule E-cadherin. Actin filaments of adjacent cells are attached to the E-cadherin­catenin complex by -actinin and vinculin. The E-cadherin­catenin complex interacts with identical molecules embedded in the plasma membrane of the adjacent cell. The plasma membranes are separated here by a relatively uniform intercellular space. This space appears clear, showing only a sparse amount of diffuse electron-dense substance, which represents extracellular domains of E-cadherin.

Honeybee Venom (Bee Venom). Aciphex.

• Are there any interactions with medications?
• What is Bee Venom?
• Are there safety concerns?
• Arthritis.
• How does Bee Venom work?
• Reducing the severity of allergic reactions to bee stings.
• Nerve pain, tendonitis, and muscle swelling (inflammation).
• Dosing considerations for Bee Venom.
• What other names is Bee Venom known by?
• Multiple sclerosis (MS).

Source: http://www.rxlist.com/script/main/art.asp?articlekey=96933

The tunica intima consists of a lining of endothelial cells that rest on a thin layer of connective tissue containing smooth muscle cells gastritis cystica profunda discount aciphex 10 mg online, occasional macrophages gastritis symptoms with back pain aciphex 20 mg lowest price, and collagen and elastic fibers. The boundary between it and the tissue below, the tunica media, is not sharply defined. The tunica media contains an abundance of smooth muscle cells (note the blue-staining nuclei) and numerous elastic fenestrated membranes (the red, wavy lamellae). The tunica adventitia, the outermost part, lacks elastic laminae, consists mainly of connective tissue, and contains the blood vessels and nerves that supply the aortic wall. Photomicrograph of a cross-section through a muscular artery in a routine H&E preparation shows that the wall of the muscular artery is also divided into the same three layers as in the elastic artery. The tunica intima consists of an endothelial lining, a small amount of connective tissue, and the internal elastic membrane. This structure has a scalloped appearance when the vessel is constricted and is highly refractile. The tunica media consists mainly of circularly arranged smooth muscle cells and collagen and elastic fibers. The nuclei of the smooth muscle cells, when contracted, have a corkscrew appearance. A well-defined external elastic membrane is not apparent in this vessel, but profiles of elastic material (arrows) are present. In larger vessels, the transport of oxygen, nutrients, and waste products to and from the lumen is supplemented by diffusion from the network of small blood vessels called vasa vasorum. It consists of small arteries that enter the vascular wall from outside the vessel and then divide into a network of arterioles and capillaries supplying the outer part of the wall. Small veins emerging from the vasa vasorum network drain the capillaries and venules into larger veins that accompany the arteries. The inner part of the vascular wall is supplied by diffusion of nutrients from the lumen. The function of vasa vasorum is to deliver nutrients and oxygen to the vascular wall and to remove waste products produced by cells residing in the wall or diffused from the lumen of the vessel. Also, a prominent internal elastic membrane becomes apparent, helping to distinguish muscular arteries from elastic arteries. The tunica intima is thinner in muscular arteries and contains a prominent internal elastic membrane. Cardiovascular System There is a strong association between the higher density of vasa vasorum in an arterial wall and the severity of atheromatous plaque formation. In some muscular arteries, the subendothelial layer is so scanty that the basal lamina of the endothelium appears to make contact with the internal elastic membrane. In this schematic diagram of a muscular artery, the cellular and extracellular components are labeled. Note the distribution of cellular components in all three tunics and the locations of external and internal elastic membranes. The relatively thick tunica media enclosed by the internal and external elastic membranes consists mainly of circularly arranged smooth muscle cells, collagen, and fine elastic fibers. The tunica intima in this preparation is indiscernible; the tunica adventitia is well defined, consisting mostly of connective tissue with collagen and elastic fibers. In muscular arteries of young adults, the tunica intima accounts for about one-sixth of the total wall thickness. In older adults, the tunica intima may be expanded by lipid deposits, often in the form of irregular "fatty streaks. Nerves and small vessels travel in the adventitia and give off branches that penetrate into the tunica media in the large muscular arteries as the vasa vasorum. Small Arteries and Arterioles Small arteries and arterioles are distinguished from one another by the number of smooth muscle cell layers in the tunica media. Cardiovascular System the tunica media of muscular arteries consists of vascular smooth muscle cells amid collagen fibers and relatively little elastic material. The smooth muscle cells possess an external (basal) lamina except at the sites of gap junctions and produce extracellular collagen, elastin, and ground substance. The tunica adventitia of muscular arteries is relatively thick and is often separated from the tunica media by a recognizable external elastic membrane. Typically, the tunica intima of a small artery has an internal elastic membrane, whereas this layer may or may not be present in the arteriole. The endothelium in both is essentially similar to endothelium in other arteries, except that at the electron microscope level, gap junctions may be found between endothelial cells and the smooth muscle cells of the tunica media. Last, the tunica adventitia is a thin, ill-defined sheath of connective tissue that blends with the connective tissue in which these vessels travel. Arterioles control blood flow to capillary networks by contraction of the smooth muscle cells. Compared with elastic arteries, the tunica adventitia of muscular arteries is relatively thick-about the same thickness as the tunica media. However, a concentration of elastic material immediately adjacent to the tunica media Arterioles serve as flow regulators for the capillary beds. In the normal relationship between an arteriole and a capillary network, contraction of the smooth muscle in the wall of an arteriole increases the vascular resistance and reduces or shuts off the blood going to the capillaries. The tunica intima of the vessel is composed of an endothelium and a very thin layer of subendothelial connective tissue (collagen fibrils and ground substance). The tunica adventitia is composed of collagen fibrils and several layers of fibroblasts (F) with extremely attenuated processes. One arteriole is seen in longitudinal section, and another is seen in cross-section.

Syndromes

• Breathing tube to help with breathing and protect the windpipe and lungs from getting eucalyptus oil into them when vomiting
• Clothing, washcloths, towels, and sheets or other items that contact infected areas should be washed in very hot (preferably boiling) water.
• Choose foods and portion sizes that promote a healthy weight.
• Heart tumors (rhabdomyoma)
• Has a seizure
• National Institute of Mental Health - www.nimh.nih.gov
• Nausea 
• Adrenal hormones
• Drug allergies
• Are having headaches more often than usual.

The epithelial cell is located on the outer (abluminal) surface of the endothelial cell gastritis diet 90 aciphex 20 mg purchase visa. Note that the endothelial cells and epithelial cells are separated by the shared basal lamina and that no collagen fibrils are present chronic gastritis message boards 10 mg aciphex order mastercard. Photomicrograph of a silver preparation revealing two longitudinally sectioned venous sinuses in the spleen. These blood vessels are surrounded by a modified basement membrane, which takes the form of a ring-like structure, much like the hoops of a barrel, rather than a continuous layer or lamina. The rings are blackened by the silver and appear as bands where the walls of the vessel have been tangentially sectioned (arrows). To the right, the cut has penetrated deeper into the vessel and shows the lumen (L). In the lower vessel, the cut rings have been sectioned in a virtually perpendicular plane, and the rings appear as a series of dots. Electron micrograph of the wall of a venous sinus, showing a longitudinally sectioned endothelial cell (EnC). The basal lamina material (asterisks) has the same homogeneous appearance as seen by electron microscopy in other sites except that it is aggregated into ring-like structures rather than into a flat layer or lamina. Moreover, its location and plane of section correspond to the silver-reactive, dot-like material in the panel above. An interwoven network of proteins provides the bases for a variety of basal lamina functions. Although morphologically all basal laminae appear similar, their molecular composition and functions are unique to each tissue. As noted, the basal lamina serves as an intermediary structure in the attachment of cells to the adjacent connective tissue. Epithelial cells are · anchored into the basal lamina by cell-to-extracellular matrix junctions, and the basal lamina is attached to underlying connective tissue by anchoring fibrils and fibrillin microfibrils. Structurally, basal and external laminae separate or isolate the connective tissue from epithelia, nerve, and muscle tissues. Connective tissue- including all of its specialized tissues, such as bone and cartilage (with the exception of adipose tissue, in that its cells possess an external lamina)-can be viewed as a single, continuous compartment. In contrast, epithelia, muscles, and nerves are separated from adjacent connective tissue by intervening basal or external laminae. This diagram shows the cellular and extracellular components that provide attachment between epithelial cells and the underlying connective tissue. On the connective tissue side of the basal lamina, anchoring fibrils extend from the basal lamina to the collagen (reticular) fibrils of the connective tissue, providing structural attachment at this site. This high-magnification electron micrograph of human skin shows the basal portion of human epithelial cells with underlying basal lamina. Anchoring filaments are responsible for attaching the basal cell membrane to the basal lamina. Filtration is well characterized in the kidney, in which the plasma filtrate must cross the compound basal laminae of capillaries and adjacent epithelial cells to reach the urinary space within a renal corpuscle. Newly formed cells or growing processes of a cell use the basal lamina that remains after cell loss, thus helping to maintain the original tissue · architecture. For example, when nerves are damaged, new neuromuscular junctions from a growing axon will be established only if the external lamina remains intact after injury. The basal laminae also allow cells to migrate under physiologic conditions but act as barriers against tumor cell invasion. Many molecules that reside in the basal lamina interact with cell surface receptors, influencing epithelial cell behavior during morphogenesis, fetal development, and wound healing by regulating cell shape, proliferation, differentiation, and motility as well as gene expression and apoptosis. For instance, the basal lamina of endothelial cells has recently been found to be involved in the regulation of tumor angiogenesis. Cell-to-Extracellular Matrix Junctions the organization of cells in epithelium depends on the support provided by the extracellular matrix on which the basal surface of each cell rests. Anchoring junctions maintain the morphologic integrity of the epithelium­connective tissue interface. The two major anchoring junctions are: · · focal adhesions, which anchor actin filaments of the cytoskeleton into the basement membrane; and hemidesmosomes, which anchor the intermedi- ate filaments of the cytoskeleton into the basement membrane. Focal adhesions are also found in other nonepithelial cells such as fibroblasts and smooth muscle cells. In general, focal adhesions consist of a cytoplasmic face to which actin filaments are bound, a transmembrane connecting region, and an extracellular face that binds to the proteins of the extracellular matrix. The main family of transmembrane proteins involved in focal adhesions is integrins, which are concentrated in clusters within the areas where the junctions can be detected. On the extracellular side, integrins bind to extracellular matrix glycoproteins, usually laminin and fibronectin. Focal adhesions play an important role in sensing and transmitting signals from the extracellular environment into the interior of the cell. Focal adhesions create a dynamic link between the actin cytoskeleton and extracellular matrix proteins. They are able to detect Focal adhesions form a structural link between the actin cytoskeleton and extracellular matrix proteins. Focal adhesions play a prominent role during dynamic changes that occur in epithelial cells. Coordinated remodeling of the actin cytoskeleton and the controlled formation and dismantling of focal adhesions contractile forces or mechanical changes in the extracellular matrix and convert them into biochemical signals. This phenomenon, known as mechanosensitivity, allows cells to alter their adhesion-mediated functions in response to external mechanical stimuli.

Usage: q.h.

During embryonic development gastritis symptoms nz buy aciphex australia, one randomly chosen X chromosome in the female zygote undergoes chromosome-wide chromatin condensation gastritis diet 6 months generic aciphex 10 mg on line, and this state is maintained throughout the lifetime of the organism. Although the Barr body was originally found in sectioned tissue, it was subsequently shown that any relatively large number of cells prepared as a smear. In cells of the oral mucous membrane, the Barr body is located adjacent to the nuclear envelope. In both sections and smears, many cells must be examined to find those whose orientation is suitable for the display of the Barr body. The second X chromosome of the female patient is repressed in the interphase nucleus and can be demonstrated in the neutrophil as a drumstick-appearing appendage (arrow) on a nuclear lobe. Granular material (pars granulosa) represents the site of initial ribosomal assembly and contains densely packed preribosomal particles. The network formed by the granular and the fibrillar materials is called the nucleolonema. The nucleolus varies in size but is particularly well developed in cells active in protein synthesis. Chromosome analysis can be performed on peripheral blood, bone marrow, tissues (such as skin or chorionic villi obtained from biopsies), and cells obtained from amniotic fluid during amniocentesis. Studies of chromosomes begin with the extraction of whole chromosomes from the nuclei of dividing cells. To obtain an image of all of the chromosomes, a mixture of different probes is used to produce different colors in each chromosome. Karyotypes labeled by this method allow cytogeneticists to perform a comprehensive analysis of changes in the number of chromosomes and chromosomal abnormalities such as additions or deletions. It is clearly visible on this color image that a part of the original chromosome 8 (aqua blue region) is now attached to chromosome 14, and a small portion of chromosome 14 (red region) is now part of chromosome 8. Note that one homolog of chromosome 15 has lost that region (no orange signal is visible). When the deletion is inherited from the mother, patients develop Angelman syndrome; when inherited from the father, patients develop Prader-Willi syndrome. The partially assembled ribosomal subunits (preribosomes) are exported from the nucleus via nuclear pores for full assembly into mature ribosomes in the cytoplasm. Nucleostemin is a newly identified protein that has been the Cell Nucleus found within the nucleolus. Nucleostemin is a p53-binding protein that regulates the cell cycle and influences cell differentiation (page 85). The presence of nucleostemin in malignant cells suggests that it could play a role in their uncontrolled proliferation (Folder 3. These viruses can use components of the nucleolus as part of their own replication process. Evidence suggests that viruses may target the nucleolus and its components to favor viral transcription and translation and perhaps alter the cell cycle to promote viral replication. The nucleolus stains intensely with hematoxylin and basic dyes and metachromatically with thionine dyes. The nuclear envelope is assembled from two (outer and inner) nuclear membranes with a perinuclear cisternal space between them. Polyribosomes are often attached to ribosomal docking proteins present on the cytoplasmic side of the outer nuclear membrane. In addition, the inner nuclear membrane contains specific lamin receptors and several lamina-associated proteins that bind to chromosomes and secure the attachment of the nuclear lamina. The nuclear lamina is formed by intermediate filaments and lies adjacent to the inner nuclear membrane. Thus, when examined in the light microscope, nucleoli appear Feulgen-negative with Feulgen-positive nucleolus-associated chromatin that often rims the nucleolus. Nuclear Envelope the nuclear envelope, formed by two membranes with a perinuclear cisternal space between them, separates the nucleoplasm from the cytoplasm. The nuclear envelope provides a selectively permeable membranous barrier between the nuclear compartment and the nuclear lamina, a thin, electron-dense intermediate filament network-like layer, resides underneath the nuclear membrane. If the membranous component of the nuclear envelope is disrupted by exposure to detergent, the nuclear lamina remains, and the nucleus retains its shape. The major components of the lamina, as determined by biochemical isolation, are nuclear lamins, a specialized type of nuclear intermediate filament (see page 63), and lamin-associated proteins. Nuclear lamina is essentially composed of lamin A and lamin C proteins that form intermediate filaments. For instance, inactivation of tumor-suppressor genes has been shown to play a role in the growth and division of cancer cells. By screening patients for mutations in these genes, much earlier detection of cancer can be accomplished. It is also now known why in some individuals, p53 mutations make their tumors resistant to radiotherapy. However, these cells will not die because of the absence of functional p53, which triggers apoptosis. This schematic drawing shows the structure of the nuclear lamina adjacent to the inner nuclear membrane. Note that the nuclear envelope is pierced by nuclear pore complexes, which allow for selective bidirectional transport of molecules between nucleus and cytoplasm. It is formed by intermediate filaments (lamins) that are arranged in a square lattice. Unlike other cytoplasmic intermediate filaments, lamins disassemble during mitosis and reassemble when mitosis ends. The nuclear lamina appears to serve as scaffolding for chromatin, chromatin-associated proteins, nuclear pores, and the membranes of the nuclear envelope. In addition, it is involved in nuclear organization, cell-cycle regulation, differentiation, and gene expression.

References

• Luostarinen L, Himanen SL, Luostarinen M, Collin P, Pirttila T. Neuromuscular and sensory disturbances in patients with well treated coeliac disease. J Neurol Neurosurg Psychiatry. 2003; 74(4):490-494.
• Morris JG Jr, Shay DK, Hebden JN, et al. Enterococci resistant to multiple antimicrobial agents, including vancomycin. Establishment of endemicity in a university medical center. Ann Intern Med. 1995;123(4):250-259.
• Peman J, Zaragoza R. Current diagnostic approaches to invasive candidiasis in critical care settings. Mycoses 2010;53(5):424-33.
• Snaith ML: ABC of rheumatology: gout, hyperuricaemia, and crystal arthritis. BMJ 310: 521, 1995.
• Peters C.A., Skoog S.J., Arant B.S. Jr et al. Summary of the AUA Guideline on Management of Primary Vesicoureteral Reflux in Children. J Urol 2010;184:1134-1344.
• Chongsrisawat V, Yimyeam P, Wisedopas N, Viravaidya D, Poovorawan Y. Unusual manifestations of gastric inflammatory fibroid polyp in a child. World J Gastroenterol 2004;10:460.
• Sarraj MA, Escalona RM, Umbers A, et al: Fetal testis dysgenesis and compromised Leydig cell function in Tgfbr3 (beta glycan) knockout mice, Biol Reprod 82(1):153n162, 2010.